{"id":8410,"date":"2012-05-05T22:29:00","date_gmt":"2012-05-05T22:29:00","guid":{"rendered":"http:\/\/www.similima.com\/?p=8410"},"modified":"2021-10-22T05:04:25","modified_gmt":"2021-10-22T05:04:25","slug":"pneumonia-and-homoeopathy","status":"publish","type":"post","link":"https:\/\/www.homeobook.com\/pneumonia-and-homoeopathy\/","title":{"rendered":"Pneumonia and Homoeopathy"},"content":{"rendered":"
Dr Beenadas<\/strong><\/p>\n Presenting complaints<\/strong><\/p>\n Cough 6 days, coughs continually for a long time. History of presenting complaints History of previous complaints<\/strong><\/p>\n Family history<\/strong><\/p>\n Personal history<\/strong><\/p>\n Physical features<\/strong><\/p>\n (a)\u00a0Generals<\/p>\n Functionals :-<\/p>\n Eliminations :-<\/p>\n (b) Response to<\/p>\n \u00a0Psychic features Regionals<\/strong><\/p>\n Physical examination<\/strong><\/p>\n Generals:-\u00a0<\/strong><\/p>\n Systemic:-\u00a0<\/strong><\/p>\n Respiratory system<\/p>\n Palpation:-<\/p>\n Percussion:-<\/p>\n Auscultation:-<\/p>\n Investigations:-<\/strong><\/p>\n 13-7-07.<\/p>\n Provisional diagnosis :<\/strong> Pneumonia.<\/p>\n Diagnosis of patient<\/strong><\/p>\n Totality of symptoms\u00a0 :-<\/p>\n Disease<\/p>\n Patient<\/p>\n Miasmatic Expressions <\/strong><\/p>\n Psora:-<\/p>\n Sycotic.<\/p>\n Evaluation of symptoms <\/strong><\/p>\n Medicines given <\/strong><\/p>\n PNEUMONIA Definition :- (clinician) Host defenses protecting lungs The entry into lower respiratory tract is protected by glottis and is cleared by coughing defense include macrophages, fibronectin, lysozymes\u00a0 lactoferrin, Ig G, defensins, cathelicidins, collectins and complement. Surfactant is bactericidal to certain pathogens and along with Ig G and fibrinectin, can oponise bacteria.<\/p>\n FACTORS IN PATHOGENISIS Microbial factors :- Micro organism develop a verity of mechanism to counteract host defense.<\/p>\n Eg:- Chlamydia pneumonia produce \u2013 ciliostatic factor;<\/p>\n Mycoplasma pneumonia\u00a0\u00a0\u00a0\u00a0\u00a0 –\u00a0\u00a0\u00a0\u00a0\u00a0 shear off cilia;<\/p>\n S.pneumonia& N.meningitidis \u2013 proteases.<\/p>\n Host factors:-Hypogammaglobulinemia, defects in phagocytosis or ciliary function, neutropaenia, functional or anatomical asplenia or a reduction in CD4 & T lymphocytes count are all host defense deficits.<\/p>\n Anatomical defects:- obstructed bronchus, bronchiectasis or sequestration of pulmonary segment etc lead to recurrent pneumonia or failure of pneumonia to resolve.<\/p>\n Pathophysiology Pathology 1.Lobar pneumonia 1) First stage \u2013 Stage of Congestion :-occurs during the first 24 hours and is characterized grossly\u00a0 by redness and a doughy consistency\u00a0 and microscopically by vascular congestion and alveolar edema. Many bacteria are present and are swept by the rapid expansion of edema fluid throughout the lobe via the Pores of Kohn. Only a few neutrophils are seen at this stage.<\/p>\n 2) 2nd<\/sup> stage \u2013 Stage of Red Hepatization.:- This term is because of the color of the lung and the similarity of its airless, noncrepitant firmness to the consistency of liver \u2013 is characterized microscopically by the presence of many erythrocytes, neutrophills, desquamated epithelial cells and fibrin in the alveolar spaces.<\/p>\n 3) 3rd<\/sup> stage \u2013 Stage of Grey Hepatization :- The ling is dry, friable and grey-brown to yellow as a consequence of a persistent fibrinopurulent exudates, a progressive disintegration of RBC and the variable presence of hemosiderin. The exudates contains macrophages, neutrophills and seldom bacteria.<\/p>\n \u00a02nd<\/sup> & 3rd<\/sup> stage lasts for 2-3 days each, with a 2-6 day duration\u00a0of maximal consolidation.<\/p>\n 4) 4th<\/sup> stage – Stage of Resolution :- characterized by erythocytic digestion of alveolar exudates, resorptin, phagositosis or coughing\u00a0 op of residual debris and restoration of the pulmonary architecture. Fibrinous inflammation may extent to\u00a0 and across the plural space, causing a rub heard by auscultation, and may lead to resolution or to organization and plural adhesions.<\/p>\n 11.Bronchopnumonia 111. Interstitial pneumonia. 1V. Miliary Pnumonia<\/strong>. Pulmonary complications in pneumonia Community accured pneumonia.(cap)\u00a0<\/strong><\/p>\n Clinical features. Physical signs :-<\/strong>\u00a0 tachypnoea, dullness\u00a0 to percussion, increased tactile and vocal fremitus, egophony, whispering pectoriloquy, crackles and pleural friction rub.<\/p>\n Single most useful clinical sign of severity of pneumonia is respiratory rate > 30\/mt. mortality rate is highest for pneumonia due to P. aeruginora, them for klebsiella, E.coli, S.aureus and Acinetobacter.<\/p>\n Worsening of co morbid conditions \u2013 DM, CCF, IHD and asthma.<\/p>\n Complication.<\/strong><\/p>\n Diagnosis<\/strong><\/p>\n 1)Chest X-Ray<\/p>\n 2) CT \u2013 detects pulmonary opacities in patient when chest X-Ray not show pneumonia or again chest X-Ray is repeated after 24-48 hours.<\/p>\n 3) Etiological Diagnosis :- blood culture, sputum strains and culture<\/p>\n Detection of antigens of pulmonary pathogens in urine:-<\/p>\n eg :- L.pneumophilia serogroup 1 antigen _ Legionnaire\u2019s disease.<\/p>\n Bacterimic pneumococcal pneumonia \u2013 S.pneumonia.<\/p>\n Serology \u2013 detection of IgM antibody or demonstration of a fourfold rise in titer of antibody.<\/p>\n Eg:- serologic tests are complement fixation, indirect immunoflouresence, ELISA.<\/p>\n Agent diagnosed are \u2013 M.pneumonias, C.pneumonias, Chlamydia psittaci, legionella, C.burnettii<\/p>\n Polymer Chain Reaction :- detection of DNA (Legionella, M.pneumonia & C.pneumonia) or RNA of microorganisms.<\/p>\n Complication:-\u00a0Pleural effusion, Lung abscess, recurrent pneumonia.<\/p>\n Aspiration pneumonia Etiology :- cause in elderly \u2013 Enterobacteriaceae, S.aureus, S.pneumoniae and H.influenza.<\/p>\n Epidemiology:- Risk Factors :- altered level of consciousness , incompetent gastro esophageal junction, elevated intragastric pressure or volume and neuromuscular disease(glottis closures)<\/p>\n Clinical Feature :- Diagnosis:-<\/strong><\/p>\n Prevention :-<\/strong> oral hygiene.\u00a0<\/strong><\/p>\n Hospital acquired (nosocomical) pneumonia. Common in medical and surgical wards, in ICU patient undergoing\u00a0 mechanical ventilation. Tubes serves direct bacterial introduction into lower respiratory tract, prevents effective coughing to clear lower respiratory secretions, damage he tracheal epithelium and allows the accumulation of oropharyngeal secretions.<\/p>\n Presence of nasotrachial or nasogastric tube increase risk of nosocomial sinusitis.<\/p>\n Etiology:-<\/strong>Common cause \u2013 S.aureus (64%) then, enterobacteriaceae.<\/p>\n Clinical features:-<\/strong>presence of a new or progressive infiltrate on CXR + at least 2 of the following \u2013 1) fever >37.8*C 2) Leukocytosis >10,000WBC\/L 3) purulent sputum.<\/p>\n Other symptoms _ dyspnoea, hypoxemia & chest pain.<\/p>\n Diagnosis :-<\/strong> endotrachial aspiration or bronchoalveolar lavage.<\/p>\n PSB( protected specimen brush) +ive if at least (10)3CFU(colony forming units) of bacteria \/ml<\/p>\n Pneumonia Rubric \u2013<\/strong>\u00a0<\/strong><\/p>\n Kent-<\/strong><\/p>\n (1) Chest-hepatizaton of lungs-<\/p>\n \u00a0\u00a0\u00a0\u00a0\u00a0 3 \u2013 phos,\u00a0 \u00a0 2 \u2013 Br, Cactus, Camph, Chel,Iod, Kali.carb, Kali.clor, Kali.iod, Lache,\u00a0 Lob, Lyco, Nux.vom, Sangu, Tubercu.<\/p>\n (2) Chest \u2013 Inflammation \u2013 Lungs<\/p>\n \u00a0\u00a0\u00a0\u00a0\u00a0 3 \u2013 Aco, Ant.tart, Ars, Bry, Car.veg, Chel, Hep, Lob, Lyco, Mer, Phos,\u00a0 \u00a0Puls, Rhus.tox, Senega, Sepia, Sul, Ver.vir<\/p>\n Boerick<\/strong><\/p>\n (1) Resp. system- Inflammation- 1) Bronchopneumonia<\/p>\n 2- Acon, Ant.tart, Ars.iod, Bry, Chel, Ipecac, Phos, Puls, Tub.<\/p>\n 1- Am.iod, Ant.ars, Ars, Bell, Bry, Iod, Kali.carb, Puls.<\/p>\n (2) Croupous pneumonia-<\/p>\n 2- Acon, Ant.tart, Bell, Brom, Bry, Chel, Iod, Kali.carb, Lyco, Phos,\u00a0 Sangu, Sul, Ver.vir.<\/p>\n 1-\u00a0 Am.iod, Ars.iod, Arn, Campher, Car.veg, Gels, Hp.sul, Ip, Lache,\u00a0 Nat.sulph, Nit.ac, Op, Rtus.tox,Tub.<\/p>\n (3) Stages of pneumonia- Congestive-<\/p>\n \u00a02 -Acon, Fer.p, Iod, Sang, Ver.vir<\/p>\n (4) Stage of Consolidation-<\/p>\n 2-Bry, Iod, Phos.<\/p>\n (5) Stage of resolution-<\/p>\n \u00a02-ant.tart, Hepar, Kali.iod, Phos, Sang, Sulp.<\/p>\n THERAPEUTICS<\/span><\/strong>\u00a0<\/strong><\/p>\n ACONITE-<\/strong> In pulmonary congestions, 1st<\/sup> stage of pneumonia. Symptoms are high fever, distinct chill, pulse- small, full, hard & tense. History of exposure to cold winds. Skin hot, dry; hard teasing and painful dry cough. Expectoration is watery, serous and\u00a0 frothy, may blood tinged. Restless tossing about ,anxiety and fear of death.<\/p>\n VER.VIR<\/strong>– Full, hard and rapid pulse, red streak through the centre of tongue. Indicated at commencement of pneumonia \u2013 violent congestion about chest preceding pneumonia. Great arterial excitement, dyspnoea, chest oppression and stomach symptoms of nausea and vomiting. High fever.<\/p>\n FER.PHOS-<\/strong> 1st<\/sup> stage of inflammation before exudation takes place. Expectoration is thin watery and blood streaked. Violent congestion of lungs- whether at onset of disease or during its course. Thus it corresponds to secondary pneumonia in the aged and debilitated. High fever oppressed and hurried breathing and bloody expectoration. Little thirst.<\/p>\n IODINE-<\/strong> 1st<\/sup> & 2nd<\/sup> stage of pneumonia,croupous form. High fever and thirst less. Rapid tendency for hepatization. Cough and dyspnoea as if chest would not expand. Sputum blood streaked.Iod 1x-3x arrests process of hepatization if given.<\/p>\n BRYONIA-<\/strong> Fever present ;sharp, stitching\u00a0 pleuritic pains. Cough is hard and dry. Sputum is scanty and rust colored. Tongue is dry. Patient wants to keep perfectly quiet. Right sided remedy. Pain in chest worse by motion, breathing and relieved by lying on right or painful side.\u00a0 For pneumonia complicated by pleurisy.<\/p>\n PHOS-<\/strong>Follows Bry and is complimentary. Lungs are hepatized. Cough with pain under sternum; mucus rales, sputa is yellowish mucus with blood streaks or rust colored.<\/p>\n HEPAR SULPH-<\/strong> After phos. as the exudates begins to soften. Cough after exposure to dry cold wind, rattling of mucus,<by cold air or drinks. Cough when any part of body is uncovered. Patient has to sit up and bend back in\u00a0asthma.<\/p>\n TUBERCULINUM-<\/strong> In lobular pneumonia rising as intercurrent from 6x-30x is important.<\/p>\n SANGUNARIA-<\/strong> Fever, burning and fullness in the upper chest. Sharp, stitching pains on right side. Dry cough, dyspnoea\u00a0 and expectoration of rust color sputum. Circumscribed redness and burning heat of the cheeks esp. afternoon. Engorgement and congestion of lungs.<\/p>\n CHELIDONIUM<\/strong>– Bilious pneumonia is indication. Stitching pain under right scapula. Cough is loose, rattling and difficult expectoration.<\/p>\n ANT.TART-<\/strong> Indicated at stage of resolution. Fine moist rales heard all over the hepatized portion of lungs. Oppression of breathing compels to sit. No expectoration. More suited in aged and in children.<\/p>\n KALI.CARB<\/strong>– Sharp stitching pain in chest, worse by motion. Affect lower part of right lung. Dyspnoea and great mucus in chest. Indicated when Ant.tart & Ip fails to raise the expectoration.<\/p>\n SULPHUR-<\/strong> In any stage of pneumonia. Slow speech, dry tongue, weakness and faintness are characteristic.Dyspnoea <12 &2 at midnight causing patient to sit-up in bed. Psoric constitutions with tendency to develop into tuberculosis.<\/p>\n LYCOPODIUM-<\/strong>Useful in delayed or partial resolution. Tightness across the chest, aching over lungs, great weakness. Threatens to run into acute phthisis.<\/p>\n IPECAC-<\/strong> Cough incessant and violent with every breath. Chest seems full of phlegm. Bubbling rales. Constriction in chest with nausea. Hoarseness. cough dry and asthmatic. Rattling cough. Fever with nausea, thirst,<\/p>\n CAL.C<\/strong> chilliness and shivering.<\/p>\n CUPRUM.MET-<\/strong> Spasm and constriction of chest. Spasmodic asthma alternating with spasmodic vomiting. Cough better\u00a0 by drinking cold water. ARB- Chest very sensitive to touch, percussion or pressure. Suffocating spells. Painless hoarseness. cough exited by tickling in the throat and accompanied by vomiting.<\/p>\n ARS.ALB<\/strong>– Respiration anxious, stertorous and wheezing. Oppression of chest on coughing, walking and on going upstairs. great anxiety. chest, shivering or great heat\u00a0 and burning in chest..<\/p>\n CARBO.VEG-<\/strong> Cough < in evening, going from a warm to cold place, in bed. Chilliness or coldness in Expectoration of green mucus or yellow pus or spitting of blood and burning pain in the chest.<\/p>\n CAUSTICUM<\/strong>– Aphonia from weakness of muscles of larynx. Hawking up of abundant mucus. Cough excited by speech and cold. Cough relieved by swallow of cold water. Cough with involuntary passage of urine.<\/p>\n NAT.SULPH-<\/strong> Sycotic pneumonia, lower lobe of right lung affected , with great soreness of chest. During cough patient has to sit up in bed and hold the chest with both hands.<\/p>\n Dr.Beenadas Dr Beenadas Presenting complaints Cough 6 days, coughs continually for a long time. Productive, white sputum. Throat irritation precedes coughing. Nausea + + +. Headache, chest pain and pain around umbilicus with cough. Cough < […]<\/a><\/p>\n<\/div>","protected":false},"author":1,"featured_media":20608,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[9925],"tags":[9933],"class_list":{"0":"post-8410","1":"post","2":"type-post","3":"status-publish","4":"format-standard","5":"has-post-thumbnail","7":"category-case-presentations","8":"tag-pneumonia-and-homoeopathy"},"yoast_head":"\n
\nProductive, white sputum. Throat irritation precedes coughing.
\nNausea + + +.
\nHeadache, chest pain and pain around umbilicus with cough.
\nCough < by slightest exertion, talking tight clothing,
\n< by cold damp weather etc.
\nDifficulty in breathing \u2013 1 week. < by coughing.
\nHoarseness \u2013 since 3 days.\u00a0<\/strong><\/p>\n
\n<\/strong>Presenting complaints started 12 days after an exposure to cold damp climate during his travel to Palghat, Coimbatore etc. Then he took some Ayurvedic Medicine, single dose on the first day itself. thereafter cough becomes productive and associated with weakness of body. Increased cough, fever and dyspnoea\u00a0 occurred two days after medicine intake. Vomited mucus once and nausea still remains. Four days back noticed edema of both legs which lasted for one\u00a0 day, during his travel. Patient feels irritable and tendency to run out of\u00a0 bed if hears voice and prefers to lye still in bed as movement brings cough.<\/p>\n\n
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\n<\/strong>Wants to be quiet. Tendency to run out of bed. Imagines he is semiconscious and talks to imaginary person. Horrible dreams make him frightful and then praying.<\/p>\n\n
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\n<\/span><\/strong>Pneumonia is the infection of alveoli, distal airways and inerstitium of lung that is manifested by\u00a0 increased weight of the lungs, replacement of normal lung\u2019s sponginess by consolidation and alveoli filled with WBC , RBC and fibrin.<\/p>\n
\n<\/strong>Pneumonia is\u00a0 constellation of symptoms and signs \u2013 fever, chill, cough, pleuritic chest pain, sputum production, hyper or hyperthermia, increased respiratory rate, dullness to percussion, bronchial breathing, egophony, crackles, wheezes, pleural friction rub with at least one opacity on chest X-Ray.\u00a0<\/strong><\/p>\n
\n<\/strong>Nasal turbinate and sharp angular turn from the naso and anterior oropharynx into posterior pharynx acts as baffles where inhaled particulate matter can impact. Ciliated cells overlying mucus layer\u00a0 of trachea, bronchi and terminal bronchioles move a mucus layer which float. Mucus consists of complex glycoproteins called mucins that trap micro-organisms.<\/p>\n
\n<\/strong>Routes of infection :- gross aspiration, micro aspiration, haematogenous spread from a distal infected site and direct spread from a contiguous infected site.<\/p>\n
\n<\/strong>Vital capacity, lung compliance, functional residual capacity and total lung capacity\u00a0 are below normal.<\/p>\n
\n<\/strong>This is manifested as 4 general patterns \u2013 1) Lobar Pneumonia 2) Broncho Pneumonia 3) Interstitial Pneumonia 4) Miliary Pneumonia.<\/p>\n
\n<\/strong>Classically involves an entire lung lobe relatively homogeneously, although in some patient a small portion of the lobe may be unaffected or at an earlier stage of involvement. Four\u00a0 stage of lobar pneumonia may exists simultaneously in the\u00a0 same lung.<\/p>\n
\n<\/strong>A\u00a0 patchy consolidation involving\u00a0 one or several lobes, usually involves the dependent lower and posterior portions of the lung \u2013 a pattern attributable to the distribution of aspirated oropharyngeal contents by gravity. The consolidated area are poorly demarcated; in some cases there is an abrupt delimitation of the pneumonia at interlobular septa. The neutrophilic exudates is centered in bronchi and bronchioles with centrifugal spread to adjacent alveoli and diminishing cellular exudates. Often there is pulmonary edema in the periphery of the lesion.<\/p>\n
\n<\/strong>Is defined by histopathologic identification of an inflammatory process predominantly involving the interstitium, including the alveolar walls and the connective tissues around the bronchovascular tree. The inflammation may be patchy or diffuse. The alveolar septa contain an inflammation of lymphocytes, macrophages and plasma cells. The alveoli contains protein rich hyaline membrane similar to that found in ARDS may line the alveolar space. Bacterial super infection of viral pneumonia also produce a mixed patter of interstitial and alveolar air space inflammation.<\/p>\n
\nNumerous discrete lesions resulting from the spread\u00a0 of the pathogen to the lungs via the bloodstream. The varying degree of immunocompramise manifests as variation in the tissue reaction.<\/p>\n
\n<\/strong>Necrotizing pneumonia, formation of abscess, vascular invasion with infarction, cavitations and extension to the pleura\u00a0 with empyema or bronchopleural fistula.\u00a0<\/strong><\/p>\n\n
\n<\/strong>Vary from mild to fulminent and fatal. Onset may be sudden, dramatic and insidious. Fever, cough (nonproductive\u00a0 or productive of purulent or rust colored sputum), pleuritic chest pain, chills or rigors, and shortness of breath are typical. Headache, nausea, vomiting, diarrhea, myalgia, arthralgia, fatigue, etc may also be present. Confusion may be important manifestation in elderly persons.<\/p>\n\n
\n<\/strong>It is due to introduction of foreign substance or objects into the lower respiratory tract. Areas involved are those that are most dependent in the supine position \u2013 posterior segment of upper lobe and superior segment of lower lobe.<\/p>\n
\n<\/strong>Gastric contents are aspirated into lungs with a consequent inflammatory response \u2013 due to aspiration of oropharyngeal flora into the lungs with consequent bacterial infection.<\/p>\n
\n<\/strong>Chemical pneumonitis which is severe, require assisted ventilation. PH of <2.5 & a gastric aspirate volume of >0.3ml\/kg(20-25ml in adults) is need to develop aspiration pneumonia.<\/p>\n\n
\n<\/strong>It is the pneumonia occurring at least 48 hours after hospital admission.<\/p>\n
\n<\/strong>Lecturer, Department of MM
\nGovt. Homeopathic Medical College. Calicut<\/p>\n","protected":false},"excerpt":{"rendered":"